Amaphupha amabi abantwana abonisa ukuphazamiseka kwengqondo

Amaphupha amabi abantwana abonisa ukuphazamiseka kwengqondo

Uphononongo olutsha lubonise ukuba abo basoloko benamaphupha amabi ebuntwaneni babo kunokwenzeka ukuba babe “nokuphazamiseka kwengqondo okubulalayo” kamva ebomini.

Uphononongo lwaqukumbela, ngokutsho kweDaily Mail, ukuba amaphupha amabi aqhubekayo ukusukela kwiminyaka esixhenxe inokuqikelela umngcipheko wesifo sengqondo esiyingozi kunye nesifo sikaParkinson kwixesha elizayo.

Kuphononongo, olwalandela abantu abangama-7000 ukususela ekuzalweni ukuya kwiminyaka engama-XNUMX ubudala, iqela leYunivesithi yaseBirmingham e-UK lathi abo babenamaphupha amabi aqhubekayo ebuntwaneni babenokuphinda kabini amathuba okuba bahlakulele isifo sengqondo esixhalabisayo kunye namathuba asixhenxe okuba nesifo sikaParkinson.

Izazinzulu zachaza ukuba ukoyikeka kwasebusuku kwasekuqaleni kobomi kunokuphazamisa ubuthongo, okuthi ngokuhamba kwexesha kukhokelela ekwandeni kweeprotheyini eziyingozi kwingqondo ezinokubangela ukuncipha kwengqondo.

Ukwenza ukuba abantwana bangabi namaphupho amabi, nokuba kungokubonelela ngokukhanya okumfiliba ebusuku, ukulandela inkqubo engaguqukiyo, okanye ukubanika into yokudlala abaza kuwagona, kunokuba neenzuzo ezinkulu zexesha elide kwiingqondo zabo.

Iingcali zenzululwazi kudala zisazi ukuba amaphupha amabi phakathi kunye nobudala anokuba luphawu olulumkisayo lokuncipha kwengqondo. Kodwa olu phononongo, lupapashwe kwijenali ye-eClinicalMedicine, lucebisa ukuba ikhonkco lidlulela ebuntwaneni

Izazinzulu zaseBirmingham zihlalutye idatha evela kwi-1958 British Birth Cohort Study.

Uphononongo lulandele idatha yabantwana abazelwe kwiveki eqala nge-3 kaMatshi ngo-1958 eNgilani, kude kube yi-2008 yokuzalwa kwabo ngo-XNUMX.

Njengenxalenye yolu phando, oomama babantwana banikezela ngolwazi malunga "namaphupha aphazamisayo kunye noloyiko lwasebusuku" kwiminyaka esixhenxe (1965) kunye ne-11 leminyaka ubudala (1969).

Abantwana abanabazali abathi banamaphupha amabi kuzo zombini ezi meko bachazwa ngokuba namaphupha amabi aqhubekayo, kwaye abantu abadala abancinci baye bajongwa de kwaba ngu-2008 ukuze kufunyaniswe ukuchaphazeleka kwengqondo, okufana nesifo sengqondo esiyingozi okanye isifo sikaParkinson.

Kubantu abangama-7000 abathathe inxaxheba kuphononongo, abantu abangama-268 (4%) baba namaphupha amabi kwasebutsheni babo, kwaye phakathi kwaba, i-17-6% yaba nokukhubazeka kwengqondo okanye isifo sikaParkinson ngexesha babefikelela kwiminyaka engamashumi amahlanu ubudala.

Ukuthelekisa, kubantu abayi-5470 abangazange babe namaphupha amabi, kuphela i-199, okanye i-3.6%, eyaba ne-dementia.

Uhlalutyo lwenziwe ngokulungelelanisa iziphumo zeminyaka yobudala, isondo, ubudala bomama ekuzalweni, inani labantakwabo, kunye nezinye izinto eziphazamisayo. Kodwa iziphumo zibonise ukuba abo banamaphupha aphazamisayo babengama-76% amathuba okuba babe nokuphazamiseka kwengqondo, kunye nama-640% amathuba okuba babe nesifo sikaParkinson. Ezi ziphumo bezifana kumakhwenkwe nakumantombazana.

Nangona kwakungacacanga ukuba kutheni amaphupha amabi enokuba luphawu lwesilumkiso sesifo sengqondo esiyingozi kunye nesifo sikaParkinson. Kodwa uphando lwangaphambili lunxulumanise oku notshintsho kwizakhiwo zengqondo ezenza umntu achaphazeleke ngakumbi kwizifo zengqondo.

Abanye baye bacebisa ukuba abo baba namaphupha amabi banomgangatho ophantsi wokulala, nto leyo enokukhokelela ekwakhiweni ngokuthe ngcembe kweeproteni ezinxulumene nesifo sengqondo esiyingozi.

Isazi se-neurologist u-Abedemi Otaiko, owayekhokela isifundo, wachaza ukuba kungenzeka ngenxa yemfuza, njengoko iprotheyini ye-PTPRJ, eyaziwayo ngokunyusa umngcipheko wokuhlala ebusuku, ihambelana nomngcipheko wesifo se-Alzheimer ekugugeni.

Ngaba siza kuthi ndlela-ntle kwi-Alzheimer's?

Kwelinye icala, yaye njengeendaba ezivuyisayo, iofisi yeendaba yasePetersburg University of Applied Sciences yaseRashiya yabhengeza iindaba ezinokuthi zenze inguqu kwezenzululwazi ukuze kupheliswe ingxaki ekhathaza abantu abaninzi, njengoko izazinzulu zaseyunivesithi ziye zenza iyeza eligcina iyeza elilondolozayo. inkumbulo kwaye iyasebenza ekulweni nesifo se-Alzheimer.

I-ofisi yaqinisekisa ukuba iimvavanyo ezenziwe kwizilwanyana zaselabhoratri zibonakalise ukusebenza kwechiza.

“Eli chiza lijolise ekunciphiseni ukulahleka konxibelelwano phakathi kweeseli, nto leyo enceda ukugcina inkumbulo. Sikholelwa ukuba isifo sika-Alzheimer siqala ngomonakalo wonxibelelwano phakathi kwee-neuron ezisengqondweni. Ukuba sinokucothisa le nkqubo, siyakulibazisa ukuqala kweempawu zesifo. ”

Ngokutsho kweofisi, iyeza lavavanywa kwizilwanyana ezazinengxaki yokukhumbula. Kwavela ukuba xa uthatha ichiza, amacandelo alo angena kumqobo wegazi-ingqondo, afikelele kwingqondo, kwaye abe nefuthe elihle kwiiseli, ezikhokelela ekubuyiselweni kwememori.

Abaphandi baceba ukufunda ichiza ngokwetyhefu, ukuguqulwa kwenguqu kunye neziphumo ebezingalindelekanga, emva koko ziya kungena kuvavanyo lweklinikhi.

Uqikelelo lukaMaguy Farah lwehoroscope yonyaka ka-2023